By De Seguir J.-A.

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Long-term risk of first recurrent stroke in the Perth community stroke study. Stroke, 29, 2491–500. J. W. (2001). Homocysteine and stroke. Current Opinion in Neurology, 14, 95–102. S. (2003). The role of endothelial dysfunction in lacunar infarction and ischaemic leukoaraiosis. Brain. 126, 424–32. Heart Protection Study Collaborative Group. (2002). MRC/BHF Heart Protection Study of cholesterol lowering with simvastatin in 20,536 high-risk individuals: a randomised placebo-controlled trial. Lancet, 360, 7–22.

It has been speculated this peri-mesencephalic subarachnoid haemorrhage may arise from a ruptured vein rather than being arterial, and possibly from a varicose vein or venous malformation (Warlow et al. 1996a). The remaining 5 per cent of spontaneous subarachnoid haemorrhages are due to a variety of other diseases including arterial dissection and various other rare conditions. Genetic studies have primarily concentrated on the genetics of aneurysmal subarachnoid haemorrhage. A major advantage in studying this condition is that new magnetic resonance angiography techniques can detect asymptomatic aneurysms in a large proportion of cases (Fig.

Sandoz, Paris. B. (2001). Anticoagulants or antiplatelet therapy for non-rheumatic atrial fibrillation and flutter. Cochrane Database Systematic Reviews, 1, CD001938. , et al. (1994). Cerebral microembolism in symptomatic and asymptomatic high-grade internal carotid artery stenosis. Neurology, 44, 615–8. A. (1999). Ethnic differences in stroke incidence. The south London Stroke register. British Medical Journal, 318, 967–71. The French Study of Aortic Plaques in Stroke Group. (1996). Atherosclerotic disease of the aortic arch as a risk factor for recurrent ischemic stroke.

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